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Vol. 4, No. 4, 2005
 
     
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COCONUT OIL
AND YOUR IMMUNE SYSTEM

by MARY ENIG


Mary Enig , PhD is the author of Know Your Fats: The Complete Primer for Understanding the Nutrition of Fats, Oils and Cholesterol (Bethesda Press 301-680-8600). She is President of the Maryland Nutritionists Association and Vice President of the Weston A Price Foundation. This piece is reprinted with the permission of Weston A. Price Foundation where the unedited version of this article appears.

* * * * * * * * * *

COCONUT OIL – THE ANATOMY OF A BAD RAP

The problems for coconut oil started four decades ago when researchers fed animals hydrogenated coconut oil that was purposefully altered to make it completely devoid of any essential fatty acids. The hydrogenated coconut oil was selected instead of hydrogenated cottonseed, corn or soybean oil because it was a soft enough fat for blending into diets due to the presence of the lower melting medium chain saturated fatty acids. The same functionality could not be obtained from the cottonseed, corn or soybean oils if they were made totally saturated, since all their fatty acids were long chain and high melting and could not be easily blended nor were they as readily digestible.

The animals fed the hydrogenated coconut oil (as the only fat source) naturally became essential fatty acid deficient; their serum cholesterol levels increased. Diets that cause an essential fatty acid deficiency always produce an increase in serum cholesterol levels as well as an increase in the atherosclerotic indices. The same effect has also been seen when other essential fatty acid deficient, highly hydrogenated oils such as cottonseed, soybean, or corn oils have been fed; so it is clearly a function of the hydrogenated product, either because the oil is essential fatty acid (EFA) deficient or because of trans fatty acids (TFA).

When unprocessed coconut oil is added to an otherwise normal diet, there is frequently no change in the serum cholesterol although some studies have shown a ‘decrease’ in total cholesterol. For example, when Ginsberg provided an Average American diet with 2-3 times more myristic acid , 4.5 times more lauric acid and 1.2 times more palmitic, serum cholesterol levels for this diet group fell approximately 3% from 177.1 mg% to 171.8 mg% during the 22 week feeding trial.

It appears from many of the research reports that the effect coconut oil has on serum cholesterol is the opposite in individuals with low serum cholesterol values and those with high serum values. We see that there may be a raising of serum total cholesterol, LDL cholesterol and especially HDL cholesterol in individuals with low serum cholesterol. On the other hand there is lowering of total cholesterol and LDL cholesterol in hypercholesterolemics as noted above.

Perhaps more important than any effect of coconut oil on serum cholesterol is the additional effect of coconut oil on the disease fighting capability of the animal or person consuming the coconut oil.

COCONUT OIL – AND THE IMMUNE SYSTEM

I would like to review the rationale for the use of coconut oil as a food that will serve as the raw material to provide potentially useful levels of anti-microbial activity in the individual.

The lauric acid in coconut oil is used by the body to make the same disease-fighting fatty acid derivative monolaurin that babies make from the lauric acid they get from their mothers’ milk.

The monoglyceride monolaurin is the substance that keeps infants from getting viral or bacterial or protozoal infections. Until just recently, this important benefit has been largely overlooked by the medical and nutrition community.

Recognition of the anti-microbial activity of the monoglyceride of lauric acid (monolaurin) has been reported since 1966. The seminal work can be credited to Jon Kabara, who, with others, observed, that certain fatty acids and their derivatives can have adverse effects on various micro-organisms: those micro-organisms that are inactivated include bacteria, yeast, fungi, and enveloped viruses.

The medium-chain saturated fatty acids and their derivatives act by disrupting the lipid membranes of the organisms. The action attributed to monolaurin is that of solubilizing the lipids and phospholipids in the envelope of the virus causing the disintegration of the virus envelope. Some of the viruses inactivated by these lipids, in addition to HIV, are the measles virus, herpes simplex virus-1 (HSV-1), vesicular stomatitis virus (VSV), visna virus, and cytomegalovirus (CMV). Many of the pathogenic organisms reported to be inactivated by these antimicrobial lipids are those known to be responsible for opportunistic infections in HIV-positive individuals. Thus, it would appear to be important to investigate the practical aspects and the potential benefit of an adjunct nutritional support regimen for HIV-infected individuals, which will utilize those dietary fats that are sources of known anti-viral, anti-microbial, and anti-protozoal monoglycerides and fatty acids such as monolaurin and its precursor lauric acid.

No one in the mainstream nutrition community seems to have recognized the added potential of anti-microbial lipids in the treatment of HIV-infected or AIDS patients. These anti-microbial fatty acids and their derivatives are essentially non-toxic to man; they are produced in vivo by humans when they ingest those commonly available foods that contain adequate levels of medium-chain fatty acids such as lauric acid. According to the published research, lauric acid is one of the best ‘inactivating’ fatty acids, and its monoglyceride is even more effective than the fatty acid alone.

LOSS OF LAURIC ACID FROM AMERICAN DIET

Increasingly, over the past 40 years, the American diet has undergone major changes. Many of these changes involve changes of fats and oils. There has been an increasing supply of the partially hydrogenated trans-containing vegetable oils and a decreasing amount of the lauric acid-containing oils. As a result, there has been an increased consumption of trans fatty acids and linoleic acid and a decrease in the consumption of lauric acid. This type of change in diet has an effect on the fatty acids the body has available for metabolic activities.

HOW MUCH LAURIC ACID IS NEEDED?

It is not known exactly how much food made with lauric oils is needed in order to have a protective level of lauric acid in the diet. Infants probably consume between 0.3 and 1 gram per kilogram of body weight if they are fed human milk or an enriched infant formula that contains coconut oil. This amount appears to have always been protective. Adults could probably benefit from the consumption of 10 to 20 grams of lauric acid per day. Growing children probably need about the same amounts as adults.

The per capita daily intake (1985) of lauric acid countries in such as the Philippines, Indonesia, and Sri Lanka, and consuming countries such as Singapore, was approximately 4.3 grams. In India, intake of lauric acid from coconut oil in the coconut growing areas (Kerala) ranges from about 12 to 20 grams per day whereas the average for the rest of the country is less than half a gram.

THE US EXPERENCE

In the United States today, there is very little lauric acid in most of the foods. During the early part of the 20th century and up until the late 1950s many people consumed heavy cream and high fat milk. These foods could have provided approximately 3 grams of lauric acid per day to many individuals. In addition, desiccated coconut was a popular food in homemade cakes, pies and cookies, as well as in commercial baked goods, and 1-2 tablespoons of desiccated coconut would have supplied 1-2 grams of lauric acid. Those foods made with the coconut oil based shortenings would have provided additional amounts.

RECOMMENDATIONS

The coconut oil industry needs to make the case for lauric acid now. It should not wait for the rapeseed industry to promote the argument for including lauric acid because of the increased demand for laurate. In fact lauric acid may prove to be a conditionally essential saturated fatty acid, and the research to establish this fact around the world needs to be vigorously promoted.

Although private sectors need to fight for their commodity through the offices of their trade associations, the various governments of coconut producing countries need to put pressure on WHO, FAO, and UNDP to recognizes the health importance of coconut oil and the other coconut products. Moreover, those representatives who are going to do the persuading need to believe that their message is scientifically correct -- because it is.

Among the critical foods and nutrition "buzz words" for the 21st century is the term "functional foods." Clearly coconut oil fits the designation of a very important functional food.

Related articles:
Teflon and your Toxicity
Retreat from Meat
Cell Phone Users Beware
Slice and Salmon Lice
The Soya Bean Conspiracy
Can Red Meat Take the Heat

 

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Does any one really believe that the Sociopathic System that controls the World cares about the health of the masses; healthy people may think better and become a real threat. Dream on corn, beets, wheat, bananas, ect. have been genetically altered. Garlic is next 2015. Coconuts? As soon as they come up with a fungal organism that kills the coconut they will have a GMO version that produces no Lauric acid along with a patent that lasts for 150 years

 


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